IdentificationofImmunodominantEpitopesontheMembraneProteinoftheSevereAcuteRespiratorySyndrome-AssociatedCoronavirus
...eofMicrobiologyandEpidemiology,Beijing100071,PeoplesRepublicofChinaABSTRACTSimilartoothercoronaviruses,themembrane(M)proteinofsevereacuterespiratorysyndrome-associatedcoronavirus(SARS-CoV)isamajortransmembraneglycoproteinwithmultiplebiologicalfunctions.Todate,limitedinformationisavailableaboutitsant...
医源资料库;在线期刊;微生物临床杂志;2005年第43卷第8期The3‘-untranslatedregionoftheSte20-likekinaseSLKregulatesSLKexpression
...】Ste20-likekinase,SLK,agerminalcenterkinasefoundinkidneyepithelialcells,signalstopromoteapoptosis.ExpressionofSLKmRNAandproteinandkinaseactivityareincreasedduringkidneydevelopmentandrecoveryfromischemicacuterenalfailure.The3‘-untranslatedregion(3‘-UTR)ofSLKmRNAcontainsmultipleadenineanduridine...
医源资料库;在线期刊;美国生理学杂志;2007年第290卷第2期MolecularandClinicalEpidemiologyofCXCR4-UsingHIV-1inaLargePopulationofAntiretroviral-NaiveIndividuals
BCCentreforExcellenceinHIV/AIDS,St.Paul‘sHospitalFacultyofMedicine,UniversityofBritishColumbia,CanadaPfizer,Inc.,NewLondon,ConnecticutObjective.WewishedtocharacterizetheepidemiologicalandclinicalcorrelatesofCXCR4-usinghumanimmunodeficiencyvirustype1(HIV-1)(“X4variants“)inacross-sectionalanalys...
医源资料库;在线期刊;传染病学杂志;2005年第191卷第15期Endothelin-1,butnotAngII,ActivatesMAPKinasesThroughc-Src–IndependentRas-Raf–DependentPathwaysinVascularSmoothMuscleCells
【摘要】Objective—Endothelin-1(ET-1)andangiotensinII(AngII)activatecommonsignalingpathwaystopromotechangesinvascularreactivity,remodeling,inflammation,andoxidativestress.Herewesoughttodeterminewhetherupstreamregulatorsofmitogen-activatedproteinkinases(MAPKs)aredifferentiallyregulatedbyET-1...
医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2007年第27卷第9期FenofibrateReducesAtherogenesisinApoE3LeidenMice
...quantify,andmechanisticallydissectantiatheroscleroticeffectsoffenofibratebesidesloweringplasmacholesterolperse.MethodsandResults-ApoE*3Leidentransgenicmicereceivedeitherahigh-cholesteroldiet(HC)orHCcontainingfenofibrate(HC+FF)resultingin52%plasmacholesterol-lowering.Inaseparatelow-cholesteroldiet(LC...
医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2006年第26卷第10期FolateintakeoftheDutchpopulationaccordingtonewlyestablishedliquidchromatographydataforfoods
...tituteNUTRIMandtheDepartmentsofHumanBiologyandEpidemiology,MaastrichtUniversity,Maastricht,Netherlands;andtheTNONutritionandFoodResearchInstitute,DepartmentofConsumerResearchandEpidemiology,Zeist,Netherlands.2SupportedinpartbytheNEVOFoundation.3AddressreprintrequeststoEJMKonings,InspectorateforHealt...
医源资料库;在线期刊;美国临床营养学杂志;2001年73卷第4期FibrinogenandfibronectinbindingcooperateforvalveinfectionandinvasioninStaphylococcusaureusexperimentalendocarditis
1DepartmentofFundamentalMicrobiology,UniversityofLausanne,1015Lausanne,Switzerland2MedicalIntensiveCareUnit,DepartmentofInternalMedicine,CentreHospitalierUniversitaireVaudois,1011Lausanne,Switzerland3ServiceofInfectiousDiseases,DepartmentofInternalMedicine,CentreHospitalierUniversitaireVaudois,1011L...
医源资料库;在线期刊;实验医学杂志;2005年第201卷第10期LuminalkidneyandintestineSLC6aminoacidtransportersofB0AT-clusterandtheirtissuedistributioninMusmusculus
...portersofB0AT-clusterandtheirtissuedistributioninMusmusculus1InstituteofPhysiologyandCenterforIntegrativeHumanPhysiology2InstituteofAnatomy,UniversityofZürich,Zürich,SwitzerlandABSTRACTTheB°transportsystemmediatestheNa+-drivenuptakeofabroadrangeofneutralaminoacidsintoepithelialcellsofsmallintesti...
医源资料库;在线期刊;美国生理学杂志;2006年第289卷第3期Foxc2IsaCommonMediatorofInsulinandTransformingGrowthFactorSignalingtoRegulatePlasminogenActivatorInhibitorTypeIGeneExpression
theDivisionofCardiovascularMedicine,DepartmentofMedicine,VanderbiltUniversityMedicalCenter,Nashville,Tenn.AbstractElevatedplasmalevelsofplasminogenactivatorinhibitortypeI(PAI-1),asignificantriskfactorofischemicheartdisease,areassociatedwithinsulinresistanceinwhichinsulinandtransforminggrowthfactor(T...
医源资料库;在线期刊;循环研究杂志;2006年第96卷第3期InvolvementofMatrixMetalloproteinaseActivityinHormone-InducedMammaryTumorRegression
...Proteolyticactivityandremodelingoftheextracellularmatrixareimportantplayersintumorprogression.However,todatetheroleoftheextracellularmatrixintumorregressionremainsunresolved.Toaddressthis,weusedaprogesterone-dependentinvivomousemammarytumorline,C4-HD,whichregressesinresponsetohormonetherapy.Withinth...
医源资料库;在线期刊;美国病理学杂志;2006年第168卷第1期